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research may have uncovered the fountain of youth for your heart 
University of Alberta Professor Jason Dyck's results indicate that the protein responsible a candidate for the drug can be inhibition for the transport of fat in the contractile cells of the heart and this drug in the heart of aging could protect. This research holds great promise for the people. Dyck hopes to the development of medicines will lead that inhibit the intake of fatty acids into the heart and to prevent and/or reverse impact of ageing on the heart muscle. An age-related decline in heart function is a risk factor for heart disease in the elderly. While many factors to a progressive age-related decline in heart function help to play heart is changes in the way of the fuels used to produce energy to produce an important role. Jason Dyck and his research team at the University of Alberta study the types of fuel used by heart mice when young and old. Young healthy heart to the usually create a balance of fat and sugar beat energy to heart to pump blood efficiently allow. As the heart of the ability to fat as an energy source but worse ages. This endangers the heart function in the elderly. Interestingly Dyck has shown at a time when the heart is less fat for energy, a protein that is responsible for the transport of fat in the contractile cells of the heart actually increases. Based on this finding, Dyck suggested that the discrepancy between fat distribution and fat to use in the heart of an accumulation of fat in the heart, which could lead to age-related decrease of the heart function. Genetically modified with a mouse in a protein deficiency is responsible for transporting fat in cells of the heart, studied Dyck these mice as you age. This genetically modified mice have no choice but to primarily use sugar as a fuel source, because you lack the protein that allows you to use fat as a primary energy source. In an exciting new knowledge Dyck showed that old GM not accumulate mice fat in their hearts as normal mice. In addition Dyck and his team showed that this old genetically modified mice old mice on a treadmill test were full of age-related decrease in heart function out performed normally and in many ways saw their hearts and run like hearts from a young mouse protected. His results indicate that the protein responsible a candidate for the drug can be inhibition for the transport of fat in the contractile cells of the heart and this drug in the heart of aging could protect. This research holds great promise for the people.Dyck hopes to the development of medicines will lead that inhibit the intake of fatty acids in the heart and to prevent and/or reverse effects of aging on the Herzmuskel.Diese study will be published in the October issue 22, 2008 of the circulation. Designed by: HostAfric.com
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